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posted by n1 on Thursday April 10 2014, @04:14AM   Printer-friendly
from the someone-has-to-do-the-research dept.

"Over a decade ago, "all human behavioral traits are heritable" was stated as the first law of behavior genetics". A new study looked at whether trust was affected by genetics.

The authors found that "genetic influences are smaller for trust, and propose that experiences with or observations of the behavior of other people shape trust more strongly than other traits".

 
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  • (Score: 0) by Anonymous Coward on Thursday April 10 2014, @10:29AM

    by Anonymous Coward on Thursday April 10 2014, @10:29AM (#29358)

    I don't want to discourage open access journals, but they sure do publish a lot of junk. Simply a reflection of today's academia, I suppose.

  • (Score: 2, Insightful) by opinionated_science on Thursday April 10 2014, @01:20PM

    by opinionated_science (4031) on Thursday April 10 2014, @01:20PM (#29424)

    Being an AC there is definitely something amusing about that statement....!

    On the topic, the study seems reasonable. The problem comes with the methods. It is very difficult to deal with opinions or feelings. Heritable traits *become* heritable, when they confer (or remove) an advantage to the organism.

    As Homo Sapiens Sapiens has its origins perhaps 2-300,000 years ago, at some point in our collected genetic past, language, and society became a positive trait.

    In the modern day it is a very complex social landscape to try and squeeze out an general concept as trust. This study used genetic controls. Probably the most interesting and suggestive finding is that "young people are more trusting".

    This strongly suggests humans have experience based adapted behaviour and less of the inherited behaviour.

    This would seem to be reasonable.

    Humans are a social animal and often it is rewarding to not standout from the crowd...

     

    • (Score: -1, Troll) by Anonymous Coward on Thursday April 10 2014, @03:49PM

      by Anonymous Coward on Thursday April 10 2014, @03:49PM (#29498)

      You are a windbag that managed to say almost nothing with all these words of yours.

      Heritable traits become heritable when it becomes heritable. There you manage to say something as opposed to absolutely nothing, except that something was wrong.

    • (Score: 1) by hellcat on Thursday April 10 2014, @05:34PM

      by hellcat (2832) on Thursday April 10 2014, @05:34PM (#29598) Homepage

      There's other points to be made here.
      First, sociobiology and Dawkins's insights (selfish gene) are quite relevant in that, technically, everything is inherited to some degree. That nurture is a cofactor isn't disputed.
      Second, oxytocin levels have been found to strongly influence 'trust' or attachment. Sorry, too busy at work to find you relevant links.
      Third, the keystone term "trait" is a land mine. There is no rigorous definition as to what constitutes a "trait." Please let me know if you find one.

      • (Score: 1) by opinionated_science on Thursday April 10 2014, @07:56PM

        by opinionated_science (4031) on Thursday April 10 2014, @07:56PM (#29675)

        The word "trait" is the neutral word from biology. It is in common usage for (Mendelian) genetics. (http://www.uni.edu/walsh/genetics.html). It did not used to be "rigorous", but with the human genome sequence in hand, it is now known at the molecular level - well *almost*, but every new genome helps... The use SNP microarrays to probe recombination blocks (sites where chromsomes tend to mix), is providing ever clear maps of what constitutes a genetic trait, although I agree the language is not unambiguous.

        The field of epigenetics has shown that it is not just genetics but environment too (via methylation, it appears).

        The problem I was trying to articulate is even with a genetic control, "trust" is a poorly defined metric. Humans in society are not the same as lab animals. There are a great many things that go into making a person, and family/society groups dominate that.

        For example, a problem with using twins is the fundamental biology that they are genotype identical, but not phenotype identical. Usually one is slightly larger....you want to bet that doesn't make a difference?

         

        • (Score: 1) by hellcat on Wednesday April 16 2014, @05:17AM

          by hellcat (2832) on Wednesday April 16 2014, @05:17AM (#32186) Homepage

          And I studied 'traits' within psychology. The word itself can be neutral, but our definition of the trait itself is influenced by many factors: personal, cultural, academic and probably social. And I find the soft science's ability to define almost anything to be rather poor.

          I agree with you wholeheartedly, and would up the ante. We *think* we have a handle on what influences (any given) trait, but the complexity of genetic expression into phenotype also increases at apparently faster rates.

          I remember learning about introns and exons - junk dna. Exons don't do anything we were taught. Chuckle. Mitochondrial dna, not important. Ha. Genetic repair machinery - infallable. Ha. Humans must have a 100,000 genes, because we're so much more complex than a mouse. Double ha.

          By the way, I'm not the one laughing here, I'm paraphrasing Mother Nature. She always has the last laugh.

          (sorry for the long delay - long overdue vacation - of two days - way overworked!)

          • (Score: 1) by opinionated_science on Wednesday April 16 2014, @11:30AM

            by opinionated_science (4031) on Wednesday April 16 2014, @11:30AM (#32253)

            wrong way around! Exons are the coding (i.e. turned into amino acids/protein) and the Introns are the spacing. Note, despite the comment misconception, many(!) biologists never really thought it was "junk DNA", since biology is necessarily quite conservative with resources. However, not knowing what is does specifically, gets communicated to the outside world as "junk". We now now it is really very important.

            There is no correlation between number of genes and organism complexity (I think this is called the C-paradox).

            The mechanism of alternative splicing is one such way in which greater complexity is obtained from few genes. The same stretch of DNA can produce different proteins, and these proteins can also be modified to function in different roles.

            Look around the natural world, and complex "preprogrammed" behaviour is everywhere. It may be something that is a "threshold" effect - you need a certain amount of "complexity" to get certain forms of intelligence, say. Just rambling...